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Their more recent operate even more disclosed that the atypical GTPase area of Fbx4 could serve as a substrate-binding motif with the SCF E3 ligase complex and bind to some globular area of TRF1 for its ubiquitination and degradation. These results present clues into the underlying mechanism for controlling TRF1, https://lennyz780mew1.blogscribble.com/profile

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